Tag: Eat

  • How We Eat vs. How We Think We Eat

    How We Eat vs. How We Think We Eat

    The so-called optimism bias may get in the way of a healthy lifestyle.

    Yes, media messages about nutrition are often confusing and inconsistent, but many Americans know what is considered a healthy diet. I mean, does anyone really think drinking brown carbonated sugar water is good for them? The issue is that they don’t appear to be translating their knowledge into action.

    Why do people have such difficulty changing their dietary behaviors? While ignorance and confusion may play a part, being motivated to change is likely much more important. Certainly, we are living in a world that pushes us to eat whatever we want, regardless of the long-term consequences. “One of the major problems in getting people to change their behaviour is the need to get them to recognise the need to change.”

    For example, if you ask people how much meat they eat—or how much greasy food, eggs, sweets, alcohol, or butter—they claim to be eating less than the average person. So, if people think they’re at less risk than others, they may dismiss advice to eat more healthfully, thinking that they already eat healthier. Is it possible that they actually are? No, people rated their own eating behavior as healthier on average, even when their actual eating habits were terrible. Because of this, perhaps campaigns promoting health need to make people aware of how badly they are eating. But when that is done, a strange thing happens. When people are challenged with the reality of what the average person actually eats, they change their answer to make themselves appear as though they’re still healthier than average.

    When people’s positive comparisons on risky behaviors are threatened, they tend to not only reduce their estimates of how frequently they engage in those behaviors—”oh, I don’t eat that much meat”—but they also minimize the significance of the behaviors. “Meat’s not that bad for you anyway.” It’s the same “personal fable” that smokers tell themselves. Studies show that smokers have a strong tendency to underestimate smoking-related risks, developing a series of illusions and false beliefs to support their choice to keep smoking.

    Why do so many people continue to light up in spite of smoking’s harm to their health? For many of the same reasons, people continue to eat unhealthy food. First, they convince themselves that they are less at risk than others who engage in the same behavior. Adding to this optimism bias, smokers also underestimate how much smoking elevates lung cancer risk, thinking two-pack-a-day smokers only have five times the risk of getting lung cancer when their actual risk is 20-fold higher, as you can see below and at 3:10 in my video Why Don’t People Eat Healthier?.Also, many smokers believe lung cancer is mainly determined by genetics.

    Many hazards related to the food we eat share this same “optimistic bias,” like heart attacks and heart disease (our number one killer), obesity, diabetes, and all the rest. People can often find quite ingenious reasons for believing that their own risk is less than others’ risk. So, maybe public health advocates need to be just as ingenious in understanding where this unrealistic optimism originated from and find ways to help people gain a more accurate picture of their own vulnerability. All sorts of work is being done trying to reduce or eliminate this bias, “but we must consider the possibility that reductions in optimistic bias might lead to reductions in self-esteem and psychological well-being,” if people start to realize just how much risk they truly face and how much they have themselves to blame.

    This reminds me of the tightrope wire health professionals have to walk, telling people how much power we all have over getting cancer. There is an oft-cited paper that calculated that we may be able to prevent approximately 90% of human cancers. Although its reference to “present trends” referred to the 1960s—when this paper was published—it still applies today, more than half a century later. “Genetic factors are not the major causes of chronic diseases.” Using identical twins to see how much disease risk was truly genetic, researchers found that out of 28 chronic diseases, cancers had the lowest genetic component—only about 10% attributable to bad genes. What runs in families is bad habits.

    But when you tell everyone the good news about how much power we have in preventing cancer, what about the people who already have it? When people are diagnosed with cancer, they often ask, “Why me? Did I do something wrong? Is this my fault?” So, you can imagine how the message of “well, yeah, kinda” could be destructive for patients or survivors. In other words, a message that is intended to empower people and promote prevention could just make cancer victims feel guilty.

    But the truth is still the truth, no matter how difficult it may be. So, what doctors have to do is try to guide patients to “switch from guilt feelings to a ‘responsibility’ approach.” They have personal control; they can make different choices from now on. Doctors need to give them a sense of agency in their lives. Better, though, to try to take those steps before you get cancer.

    Doctor’s Note

    For more on personal responsibility, see Why You Should Care About Nutrition and Taking Personal Responsibility for Your Health.



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  • Hidden Gout Triggers in Purine Foods You Eat Every Day

    Hidden Gout Triggers in Purine Foods You Eat Every Day

    High uric acid levels are a growing concern as more people experience painful gout attacks linked to their everyday diets. This article explains how certain purine foods act as gout triggers and how simple nutrition changes can help manage the condition.

    High Uric Acid and Gout

    High uric acid, medically called hyperuricemia, occurs when the body produces too much uric acid or cannot effectively remove it through the kidneys. Excess uric acid can form sharp crystals in the joints, leading to gout, a form of inflammatory arthritis known for sudden, intense pain. An objective look at research and clinical guidance shows that diet, especially purine-rich foods, plays a major role as a modifiable risk factor for gout triggers.

    What Is High Uric Acid?

    Uric acid is a waste product formed when the body breaks down purines, which are natural compounds found in both body tissues and many foods. When uric acid levels become too high, either due to overproduction or poor excretion, it can build up in the blood and eventually crystallize in joints and surrounding tissues. Persistent high uric acid increases the likelihood of recurrent gout attacks and may also contribute to kidney stones and other complications.

    How Do Purine Foods Trigger Gout?

    Purine foods are not inherently harmful, but excessive intake can overload the body’s ability to process and excrete uric acid. When purine-rich foods are digested, they break down into uric acid; if intake is consistently high, blood levels can rise beyond the body’s safe threshold. Animal-based purine foods, such as certain meats and seafood, tend to be stronger gout triggers than plant-based purine sources, which usually have a milder impact on gout risk.

    Organ Meats and Red Meat: High-Purine Protein Sources

    Organ meats rank among the highest purine foods and are strongly associated with elevated uric acid levels. Regular consumption of liver, kidneys, and similar cuts can significantly increase gout risk, so many clinical diet guidelines recommend avoiding or strictly limiting them for anyone with gout or high uric acid.

    Red meat such as beef, lamb, and pork contains moderate to high levels of purines and can also act as gout triggers when eaten in large portions or on a daily basis.

    A more objective, moderate approach focuses on reducing both portion size and frequency rather than insisting on an entirely meat-free diet. For example, replacing large daily servings of red meat with smaller portions a few times a week and incorporating plant-based proteins on other days can help lower overall purine load, according to the World Health Organization.

    This strategy allows better management of high uric acid while keeping meals realistic and sustainable for most people.

    Do Alcohol and Beer Increase Uric Acid?

    Alcohol is one of the most consistent lifestyle-related gout triggers, with beer standing out as a particular concern. Beer contains purines from brewer’s yeast and also interferes with the kidneys’ ability to get rid of uric acid, which can result in elevated levels and more frequent gout flares. Spirits and wine can also contribute when consumed in excess, but beer tends to be more closely linked with gout in observational studies.

    From an objective perspective, the safest approach for individuals with high uric acid or gout is to limit or avoid alcohol, especially during or near a flare. For those who choose to drink, moderating intake and avoiding binge patterns is essential to reduce the risk of triggering acute gout attacks. Combining alcohol reduction with other dietary changes multiplies the benefit for overall uric acid management.

    Are Sugary Drinks and Fructose Gout Triggers?

    Sugary drinks, particularly those sweetened with high-fructose corn syrup, are another major dietary factor that can exacerbate high uric acid. Fructose metabolism in the liver promotes uric acid production, and frequent consumption of sodas, energy drinks, and sweetened juices has been linked to higher gout risk.

    Unlike glucose, fructose directly raises uric acid in a way that makes these beverages especially problematic for susceptible individuals, as per the Centers for Disease Control and Prevention.

    Replacing sugary drinks with water, sparkling water, or unsweetened tea can significantly reduce this source of gout triggers. Reading labels for high-fructose corn syrup and limiting processed snacks, desserts, and condiments that contain it further supports better uric acid control. These changes also have broader metabolic benefits, including weight management and improved blood sugar control.

    What Foods Help Lower Uric Acid Naturally?

    Certain foods and dietary patterns may help reduce high uric acid levels and lower the frequency of gout attacks. Low-fat dairy products such as skim milk and yogurt have been associated with a lower risk of gout, potentially because they promote uric acid excretion. High-fiber whole grains and a variety of vegetables support healthy weight management and better overall metabolic function, both of which indirectly help with uric acid control.

    Cherries and other deeply colored fruits are often highlighted for their potential anti-inflammatory and uric-acid-lowering effects. While they are not a cure, including cherries, berries, and other fruits as part of a balanced diet may help reduce gout flares for some individuals. Overall, a diet rich in plant-based foods, low-fat dairy, and moderate protein appears to be favorable for gout management.

    What Is the Best Diet Plan for Gout?

    Objectively, no single perfect diet exists for everyone with gout, but several evidence-informed patterns are consistently recommended. A low-purine diet that limits organ meats, certain seafoods, large portions of red meat, alcohol, and sugary drinks forms the foundation of many gout management plans.

    Variations of the Mediterranean or DASH-style diets, which emphasize vegetables, fruits, whole grains, legumes, nuts, and healthy fats, also align well with gout-friendly principles.

    Hydration, weight management, and regular physical activity are important companions to changes in purine foods. Drinking enough water helps the kidneys flush out uric acid, while losing excess weight can improve both uric acid levels and overall joint health. This comprehensive lifestyle approach addresses not just gout triggers but broader cardiovascular and metabolic risks too.

    When to See a Doctor for High Uric Acid or Gout

    Medical evaluation is essential when gout symptoms appear, especially during a first attack or if pain and swelling are severe. Classic signs include sudden, intense joint pain (often in the big toe), redness, warmth, and tenderness that can make even light contact unbearable. Ignoring repeated flares can allow joint damage and tophi (crystal deposits under the skin) to develop over time.

    Anyone with known high uric acid or suspected gout should seek professional advice to confirm the diagnosis and create an appropriate treatment plan. A healthcare provider can recommend blood tests, imaging, medications, and personalized dietary guidance based on individual risk factors. Prompt intervention supports better long-term outcomes and reduces the chance of permanent joint or kidney complications.

    Frequently Asked Questions

    1. Can high uric acid levels be present without any gout symptoms?

    Yes, many people have high uric acid levels (hyperuricemia) without experiencing any joint pain or gout attacks. In these cases, the condition is often discovered incidentally during routine blood tests.

    Even without symptoms, persistently high uric acid can increase long-term risk of gout, kidney stones, and kidney disease. Healthcare providers may recommend lifestyle changes, and sometimes medication, based on overall risk factors rather than symptoms alone.

    2. Does intermittent fasting affect uric acid and gout risk?

    Intermittent fasting can temporarily increase uric acid levels, particularly in the early stages, because fasting and rapid weight loss may promote breakdown of body tissues, releasing purines into the bloodstream. For some individuals prone to gout, this can potentially trigger flares if fasting is extreme or poorly managed.

    However, gradual, well-planned weight loss with adequate hydration and balanced meals during eating windows can improve metabolic health overall and may help reduce gout risk over time. Anyone with a history of gout should discuss fasting plans with a healthcare professional before making major changes.

    3. How does dehydration influence gout attacks?

    Dehydration concentrates uric acid in the blood because there is less fluid available for the kidneys to filter and excrete it. When uric acid becomes more concentrated, the likelihood of crystal formation in the joints increases, which can precipitate a gout flare.

    Staying well hydrated helps the kidneys flush out uric acid more efficiently and is a simple, non-pharmacologic strategy to support gout management. People with gout are often advised to drink water regularly throughout the day and to increase intake in hot weather or during illness.

    4. Are supplements like vitamin C or cherry extract helpful for high uric acid?

    Some research suggests that vitamin C might modestly lower uric acid levels by enhancing kidney excretion, and cherry products (juice, extract, or whole cherries) may help reduce the frequency of gout flares due to their antioxidant and anti-inflammatory properties. These effects are usually mild and work best as part of a broader lifestyle strategy rather than as stand-alone treatments.



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  • Are Raw Mushrooms Safe to Eat? 

    Are Raw Mushrooms Safe to Eat? 

    Microwaving is probably the most efficient way to reduce agaritine levels in fresh mushrooms.

    There is a toxin in plain white button mushrooms called agaritine, which may be carcinogenic. Plain white button mushrooms grow to be cremini (brown) mushrooms, and cremini mushrooms grow to be portobello mushrooms. They’re all the very same mushroom, similar to how green bell peppers are just unripe red bell peppers. The amount of agaritine in these mushrooms can be reduced through cooking: Frying, microwaving, boiling, and even just freezing and thawing lower the levels. “It is therefore recommended to process/cook Button Mushroom before consumption,” something I noted in a video that’s now more than a decade old.

    However, as shown below and at 0:51 in my video Is It Safe to Eat Raw Mushrooms?, if you look at the various cooking methods, the agaritine in these mushrooms isn’t completely destroyed. Take dry baking, for example: Baking for ten minutes at about 400° Fahrenheit (“a process similar to pizza baking”) only cuts the agaritine levels by about a quarter, so 77 percent still remains.

    Boiling looks better, appearing to wipe out more than half the toxin after just five minutes, but the agaritine isn’t actually eliminated. Instead, it’s just transferred to the cooking water. So, levels within the mushrooms drop by about half at five minutes and by 90 percent after an hour, but that’s mostly because the agartine is leaching into the broth. So, if you’re making soup, for instance, five minutes of boiling is no more effective than dry baking for ten minutes, and, even after an hour, about half still remains.

    Frying for five to ten minutes eliminates a lot of agartine, but microwaving is not only a more healthful way to cook, but it works even better, as you can see here and at 1:39 in my video. Researchers found that just one minute in the microwave “reduced the agaritine content of the mushrooms by 65%,” and only 30 seconds of microwaving eliminated more than 50 percent. So, microwaving is probably the easiest way to reduce agaritine levels in fresh mushrooms. 
    My technique is to add dried mushrooms into the pasta water when I’m making spaghetti. Between the reductions of 20 percent or so from the drying and 60 percent or so from boiling for ten minutes and straining, more than 90 percent of agaritine is eliminated.

    Should we be concerned about the residual agaritine? According to a review funded by the mushroom industry, not at all. “The available evidence to date suggests that agaritine from consumption of…mushrooms poses no known toxicological risk to healthy humans.” The researchers acknowledge agartine is considered a potential carcinogen in mice, but then that data needs to be extrapolated to human health outcomes.

    The Swiss Institute of Technology, for example, estimated that the average mushroom consumption in the country would be expected to cause about two cases of cancer per one hundred thousand people. That is similar to consumption in the United States, as seen below and at 3:00 in my video, so “one could theoretically expect about 20 cancer deaths per 1 x 106 [one million] lives from mushroom consumption.” In comparison, typically, with a new chemical, pesticide, or food additive, we’d like to see the cancer risk lower than one in a million. “By this approach, the average mushroom consumption of Switzerland is 20-fold too high to be acceptable. To remain under the limit”—and keep risk down to one in a million—“‘mushroom lovers’ would have to restrict their consumption of mushrooms to one 50-g serving every 250 days!” That’s about a half-cup serving once in just over eight months. To put that into perspective, even if you were eating a single serving every single day, the resulting additional cancer risk would only be about one in ten thousand. “Put another way, if 10,000 people consumed a mushroom meal daily for 70 years, then in addition to the 3000 cancer cases arising from other factors, one more case could be attributed to consuming mushrooms.” 
    But, again, this is all based “on the presumption that results in such mouse models are equally valid in humans.” Indeed, this is all just extrapolating from mice data. What we need is a huge prospective study to examine the association between mushroom consumption and cancer risk in humans, but there weren’t any such studies—until now.

    Researchers titled their paper: “Mushroom Consumption and Risk of Total and Site-Specific Cancer in Two Large U.S. [Harvard] Prospective Cohorts” and found “no association between mushroom consumption and total and site-specific cancers in U.S. women and men.”

    Eating raw or undercooked shiitake mushrooms can cause something else, though: shiitake mushroom flagellate dermatitis. Flagellate as in flagellation, whipping, flogging. Below and at 4:48 in my video, you can see a rash that makes it look as if you’ve been whipped.

    Here and at 4:58 in my video is another photo of the rash. It’s thought to be caused by a compound in shiitake mushrooms called lentinan, but because heat denatures it, it only seems to be a problem with raw or undercooked mushrooms.

    Now, it is rare. Only about 1 in 50 people are even susceptible, and it goes away on its own in a week or two. Interestingly, it can strike as many as ten days after eating shiitake mushrooms, which is why people may not make the connection. One unfortunate man suffered on and off for 16 years before a diagnosis. Hopefully, a lot of doctors will watch this video, and if they ever see a rash like this, they’ll tell their patients to cook their shiitakes.



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  • Is There a Limit to How Many Lychee Fruit We Should Eat? 

    Is There a Limit to How Many Lychee Fruit We Should Eat? 

    There is a toxin in lychee fruit that can be harmful, but is it harmful only under certain circumstances?

    Lychee fruits have been widely used in many cultures for the folk medicine treatment of everything from farting to testicular swelling. (Arsenic, mercury, and lead are also included in many “traditional” remedies.) Lychees have also apparently “been shown to exhibit numerous health benefits,” but the studies cited include ones like this: “Protective Effect of a Litchi [Lychee]…-Flower-Water-Extract on Cardiovascular Health in a High-Fat/Cholesterol-Dietary Hamsters.” What are we supposed to get from that? We don’t eat lychee flowers…and we aren’t hamsters. Hard to argue with this, though: “Flavor is sweet, fragrant, and delicious,” which is why I love them so much. I then saw this: “A child-killing toxin emerges from shadows. Scientists link mystery deaths…to the consumption of lychees.”

    In Vietnam, it’s called “nightmare” encephalitis. There were unexplained outbreaks in children coinciding with lychee harvesting. Children go to bed feeling fine, but they wake up the next morning “seriously ill with brain function derangement and seizures”—if they wake up at all. The same in India, killing up to nearly two out of three kids affected in some places. We’re talking about thousands of kids, so it became “one of the most pressing public health emergencies in India.” It was one of the “three long-standing mystery diseases listed in Wikipedia” and remained a mystery for more than two decades.

    All clinical samples were negative for known brain viruses. So, some investigators thought it was caused by an unknown virus, while others thought it might have been due to the pesticides used in the orchards. All we knew was that it seemed to coincide with the lychee harvest. So, might the fruits have attracted fruit bats, then mosquitos could have fed on the infected bats and transferred some new virus from bats to people? Maybe, but why would toddlers and babies be mostly spared? Mosquitoes bite infants, too.

    So, were kids swapping spit with the fruit bats by eating half-eaten fruits? “The investigators noted colonies of fruit-eating bats and the tendency of children eating fruits to fall to the ground and suggested the possibility of a bat virus (through saliva contamination on fruits) as a cause of the disease.” Or maybe it was because it was summertime, and they were all just getting heat stroke? Maybe, but why weren’t the pesticides or the heat affecting adults, too?

    One of the clues that finally helped investigators tease out the mystery was that the children consistently had low blood sugars—in some cases, fatally low blood sugars. That kind of sounds like “Jamaican vomiting sickness.” Two children “were perfectly well” when they went to bed, but, by “the next morning, they started to vomit and were weak,” then unconscious, then both dead within 48 hours. That was all due to eating unripe ackee fruit, which contains a toxin known as hypoglycin, which prevents our liver from churning out blood sugar all night long to keep our brains alive while we sleep. Ackee is a member of the soapberry family, just like the lychee is. Aha!

    As I discuss in my video Lychee Fruit and Hypoglycin: How Many Are Too Many?, Muzaffarpur is a leading lychee producer, and experts at the National Center for Litchi claim they “completely refuted” the lychee link. Nevertheless, independent researchers found it: Lychee fruit contains methylene cyclopropyl-glycine, nearly the same hypoglycin toxin “present in ackee fruits, popular in Jamaica.”

    So, in the setting of malnourished children who already have depleted energy stores in their livers “(due to missed meals and poverty-related starvation),” low blood sugar sets in, and, due to the excessive consumption of lychee fruits, the production of new energy is blocked, and the trouble starts. “It is a social tragedy that children have to die in the 21st century due to…hypoglycemia [low blood sugar], which is an easily treatable condition and involves minimal costs.” It’s just as tragic that hungry children are forced to binge on lychees falling on the ground to get a meal. It’s like something out of Grapes of Wrath.

    The happy ending, though, is that rather than just focusing on better treatments, local public health workers instead sought to treat the cause by educating people “that no child should go to bed at night without eating a cooked meal and for parents to restrict children eating litchis in the evening to none or very few.” Thankfully, “by applying these recommendations, the disease incidence had been dramatically reduced and death almost completely prevented.” In hindsight, it appears China had already started warning citizens about the dangers of lychees a decade earlier, but word had apparently not gotten around. 

    What are the implications in the West? In the United States, the Food and Drug Administration tried to protect people against poisoning with this toxin (which is not destroyed by heating) by mandating that canned ackee fruits coming into the country test below a certain level, but there are no such regulations when it comes to importing lychees. “Fortunately, the high cost of these imported fruits and the likelihood that [they] would be eaten in small quantities by well-nourished consumers, suggests there is little reason for concern in the USA.” That’s quite an assumption. Small quantities? You don’t know how I eat lychees. I used to sneak big bags of them—pounds of them—into movie theaters to snack on during the film. How many are too many to eat?

    In a series of a few hundred poisoning cases, people reported eating 300 grams to a kilogram of lychee fruits. Each lychee is about 10 grams, so that’s 30 to 100 fruits. Most of the cases were children, though, so we can probably safely say 30 to 100 lychees are too many at one time for kids. What about adults? In a self-experiment, a researcher ate some lychees and measured the hypoglycin levels in his blood and urine, which stayed below the levels seen in the affected children. He ate 5 grams of canned lychee for each kilogram of his body weight, equivalent to about 45 lychee for the average American male, and didn’t suffer any ill symptoms. 

    What a fascinating story! A lot of research went into just this one topic, but it was all news to me, so I wanted to share it with you.

    In general, Is Canned Fruit as Healthy? And, given the sugar content, How Much Fruit Is Too Much? Check out the videos to find out. 



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  • No Safe Way To Eat It

    No Safe Way To Eat It

    When you have a curious toddler around, every corner of your home seems to pose a potential danger. From choking on small objects to accidental ingestion or poisoning, parents are constantly on alert. But what if danger is lurking in the form of something as innocent as a popular snack?

    A pediatrician is now sounding the alarm about popcorn, labeling it a dangerous choking hazard for young children and urging parents to reconsider serving it to their little ones.

    According to Dr. Niamh Lynch, a pediatrician popular on social media for her valuable health advice, there is no safe way to feed popcorn to kids. In a recent TikTok video, she bluntly stated, “What do I recommend as the safest way to feed a toddler popcorn? I don’t. It’s really dangerous.”

    The warning might come as a shocking revelation for many, especially since popcorn is a staple snack at so many kids’ parties, movie nights, and family gatherings.

    In the video, Dr. Lynch explained how popcorn poses a serious choking hazard for toddlers. She explained that popcorn can easily be inhaled into a child’s tiny airways, which are about the width of a little finger. Using diagrams, she showed how easily a toddler’s airway can become obstructed.

    Dr. Lynch also stressed that it’s not just whole pieces of popcorn that are dangerous; even small popcorn “particles” can pose a significant risk.

    “If they are aspirated or breathed in, they can settle down in the lungs and cause significant infection. So if a toddler aspirates into the airway, unfortunately it can cause them to pass away – and if they inhale little particles it can cause a really serious infection. So no, popcorn is too dangerous and it’s not recommended by pediatricians,” she said in the video.

    The U.S Center for Disease Control and Prevention also cautions against serving popcorn for kids. “Avoid serving children under 4 years old popcorn, spoonful of nut butter, whole grapes and cheese cubes, as these are common choking hazards. Always supervise children while they eat,” the CDC warns.

    To prevent choking, the CDC advises parents and caregivers to always keep children upright while eating, as this position helps reduce the risk of food blocking their airways. They also recommend avoiding feeding children in strollers, where it is difficult to monitor their eating habits. Mealtimes should be calm and focused. Also, caregivers should always closely watch what children put in their mouths.



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  • Planning To Eat Clean In The New Year? Your Obsession With a Healthy Diet Shouldn’t Turn Into Eating Disorder

    Planning To Eat Clean In The New Year? Your Obsession With a Healthy Diet Shouldn’t Turn Into Eating Disorder

    With the New Year just around the corner, so are resolutions for a healthier year. If you have already committed to eating clean in the coming year, be cautious—obsessing over healthy eating can take a toll on your mental health and potentially lead to eating disorders.

    Orthorexia nervosa (ON) is an obsession with eating “pure” foods to the point that it can take over your social life and mental well-being. Individuals may start organizing their entire schedule around strict dietary rules, skipping social events like Christmas parties or family dinners, and feel stressed or anxious when their eating routines are disrupted.

    Many people may fall into the trap of extreme dieting that cuts out carbohydrates, proteins, and essential vitamins, all in the name of healthy eating. However, this approach can put the body at risk of nutrient deficiencies, leading to symptoms such as hair loss, brittle nails, missed menstrual cycles, and constant fatigue.

    If this obsession with healthy eating goes unchecked, it can escalate into more serious problems, potentially leading to clinical eating disorders such as anorexia or bulimia.

    In a groundbreaking study, researchers explored how healthy eating addiction and extreme beauty ideals among fashion models can trigger eating and body image disorders. Their findings published in Eating and Weight Disorders – Studies on Anorexia, Bulimia, and Obesity, revealed that while 95% of the participants both models and the control group had positive emotions associated with healthy eating, over 35% of female models exhibited signs of ON and over 20% in the control group. The survey also revealed a troubling trend regarding the body mass index (BMI) of the fashion models, with a significant 88.7% of them falling below the underweight threshold.

    For those looking to adopt a healthy lifestyle in the New Year, Dr. Nikolett Bogár, a PhD student researching eating disorders at the Institute of Behavioral Sciences at Semmelweis University, recommends focusing on a long-term, balanced diet while recognizing that eating is not just about nutrition but also a social and cultural experience. She advises against categorizing foods as strictly good or bad.

    “Aim for a long-term, balanced diet rather than an ultra-clean one in January. Occasionally indulging in chocolates or holiday treats should be part of your diet – without guilt,” Dr. Bogár, advises.

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  • Eat Quinoa and Lower Triglycerides?

    Eat Quinoa and Lower Triglycerides?

    How do the nutrition and health effects of quinoa compare to other whole grains?

    “Approximately 90% of the world’s calories are provided by less than one percent of the known 250,000 edible plant species.” The big three are wheat, corn, and rice, and our reliance on them may be unsustainable, given the ongoing climate crisis. This has spurred new interest in “underutilized crops,” like quinoa, which might do better with drought and heat.

    Quinoa has only recently been introduced into the Northern Hemisphere, but humans have been eating quinoa for more than 7,000 years. Is there any truth to its “superfood” designation, or is it all just marketing hooey?

    Quinoa is a “pseudograin,” since the plant it comes from isn’t a type of grass. “Botanically speaking quinoa is an achene, a seed-like fruit with a hard coat,” and it has a lot of vitamins and minerals, but so do all whole grains. It also has a lot of protein. As you can see below and in a series of graphs starting at 1:05 in my video Benefits of Quinoa for Lowering Triglycerides, quinoa has more protein than other grains, but since when do we need more protein? Fiber is what we’re sorely lacking, and its fiber content is relatively modest, compared to barley or rye. Quinoa is pretty strong on folate and vitamin E, though, and it leads the pack on magnesium, iron, and zinc. So, it is nutritious, but when I think superfood, I think of something with some sort of special clinical benefit. Broccoli is a superfood, strawberries are a superfood, and so is garlic, but quinoa? Consumer demand is up, thanks in part to “perceived health benefits,” and it has all sorts of purported benefits in lab animals, but there have been very few human studies. 

    The first trial was a before-and-after study of quinoa granola bars that showed drops in triglycerides and cholesterol, as you can see below and at 1:53 in my video, but it didn’t have a control group, so we don’t know how much of that would have happened without the quinoa. The kind of study I want to see is a randomized controlled trial. When researchers gave participants about a cup of cooked quinoa every day for 12 weeks, they experienced a 36 percent drop in their triglycerides. That’s comparable to what one gets with triglyceride-lowering drugs or high-dose fish oil supplements.

    Which is better, regular quinoa or red quinoa? As you can see in the graph below and at 2:22 in my video, the red variety has about twice the antioxidant power, leading the investigators to conclude that red quinoa “might…contribute significantly to the management and/or prevention of degenerative diseases associated with free radical damage,” but it’s never been put to the test. 

    What about black quinoa? Both red and black quinoa appear to be equally antioxidant-rich, both beating out the more conventional white variety, as you can see in the graph below and at 2:46 in my video

    The only caveat I could find is to inform your doctor before your next colonoscopy or else they might mistake quinoa for parasites. As reported in a paper, a “colonoscopy revealed numerous egg-like tan-yellow ovoid objects, 2 to 3 mm in diameter, of unclear cause,” but they were just undigested quinoa.

    For more on the superfoods I mentioned, check the related posts below.

    Isn’t fish oil important to heart health? Find out in my video Is Fish Oil Just Snake Oil?.



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