Health Journal Online

Tag: AutoImmune

  • Autoimmune Disease Causes and Why the Immune System Attacks the Body

    Autoimmune Disease Causes and Why the Immune System Attacks the Body

    Autoimmune diseases occur when the body’s defense system mistakenly targets its own healthy tissues, and understanding autoimmune disease causes is key to making sense of this process.

    Instead of attacking only viruses, bacteria, and other invaders, the immune system becomes confused and identifies normal cells as threats. This misfire can damage joints, glands, organs, and other tissues, leading to a range of conditions such as type 1 diabetes, rheumatoid arthritis, and lupus.

    How the Immune System Normally Works

    In a healthy person, the immune system acts like a security team that distinguishes between the body’s own cells and foreign invaders.

    White blood cells and antibodies recognize markers on pathogens and respond by neutralizing or destroying them. This recognition process usually protects the body from infection while leaving healthy tissues unharmed.

    What Happens in an Autoimmune Disease?

    In autoimmune diseases, this recognition system breaks down. The immune system creates autoantibodies that target the body’s own cells or activates immune cells that attack normal tissues as if they were dangerous.

    Over time, this can cause chronic inflammation, pain, and organ dysfunction. Some conditions focus on one organ, while others affect multiple systems.

    How Many Autoimmune Diseases Are There?

    Experts estimate that there are more than 80 autoimmune diseases. Some, like psoriasis or Hashimoto’s thyroiditis, are relatively common, while others are rare and harder to recognize. Because symptoms often overlap, these conditions are grouped as autoimmune disorders driven by similar immune system errors.

    What Are the Main Autoimmune Disease Causes?

    Autoimmune disease causes are complex and usually involve several factors rather than a single trigger. Genetics, environmental exposures, infections, hormones, and lifestyle all appear to influence risk. No single factor explains every case; instead, risk comes from interactions between a person’s underlying susceptibility and their environment.

    Is Autoimmune Disease Genetic or Environmental?

    Genetics play a strong role in autoimmune disease causes. People with a family history of conditions like lupus, multiple sclerosis, or celiac disease are more likely to develop an autoimmune disorder.

    However, many individuals with risk genes never develop disease, suggesting that environmental triggers—such as infections, certain drugs, or pollutants—may “switch on” disease in those who are genetically predisposed.

    Can Infections, Stress, and Lifestyle Trigger Disease?

    Some infections may trigger autoimmune diseases through mechanisms like molecular mimicry, where parts of a virus or bacterium resemble the body’s own proteins. When the immune system attacks the infection, it may also begin targeting similar-looking tissues, according to Harvard Health.

    Stress and physical trauma can alter immune and hormone balance and may contribute to symptom onset or flare-ups, especially in those already at risk. Lifestyle factors such as smoking, obesity, poor diet, and exposure to toxins can amplify inflammation and appear to influence both the development and severity of autoimmune diseases.

    Are Autoimmune Diseases More Common in Women?

    Many autoimmune diseases are more common in women, especially during their reproductive years. Hormonal differences, including the effects of estrogen on the immune system, may help explain this pattern. Researchers continue to study how sex hormones interact with genes and environmental factors.

    Does Autoimmune Disease Run in Families?

    Autoimmune conditions often appear in families, even when relatives have different diagnoses. One person might have type 1 diabetes, another thyroid disease, and another lupus. This suggests that people may inherit a general tendency toward autoimmunity rather than a single specific condition.

    Organ-Specific vs Systemic Autoimmune Diseases

    Autoimmune diseases can be organ-specific or systemic. Organ-specific conditions mainly target one tissue or gland, such as the thyroid in Graves’ disease or the pancreas in type 1 diabetes. Systemic diseases, like lupus or vasculitis, affect multiple organs and often cause more widespread symptoms.

    Common Autoimmune Diseases and Early Signs

    Well-known autoimmune diseases include rheumatoid arthritis (joints), systemic lupus erythematosus (multiple organs), type 1 diabetes (pancreas), multiple sclerosis (nervous system), Hashimoto’s thyroiditis and Graves’ disease (thyroid), celiac disease (intestine), and inflammatory bowel diseases.

    Early symptoms are often subtle and nonspecific: fatigue, joint or muscle pain, low-grade fever, skin rashes, digestive issues, hair loss, or numbness. Because these signs resemble many other conditions, autoimmune disease can be difficult to recognize early, as per Cleveland Clinic.

    How Are Autoimmune Diseases Diagnosed?

    Diagnosis usually combines medical history, physical exam, and targeted tests. Blood tests can detect autoantibodies and markers of inflammation.

    Imaging studies such as X-rays or MRI scans may reveal joint or organ damage, and biopsies can confirm immune-related injury. Because symptoms overlap with many other illnesses, it may take time and specialist referrals to reach a clear diagnosis.

    How Are Autoimmune Diseases Treated?

    Most autoimmune diseases are chronic and cannot currently be cured, but many can be controlled. Treatment aims to reduce inflammation, calm the overactive immune response, protect organs, and relieve symptoms.

    Common medications include anti-inflammatory drugs, immunosuppressants, and biologic therapies that target specific immune pathways involved in autoimmune disease causes. Short-term corticosteroids may be used to manage flares, while disease-modifying drugs aim to limit long-term damage.

    Can Lifestyle Changes Help?

    Lifestyle changes can support medical treatment and improve quality of life. Regular physical activity, balanced nutrition, adequate sleep, and stress management can help regulate immune function and may reduce flare frequency for some individuals.

    Avoiding smoking and limiting exposure to known triggers can further support disease control.

    Prevention, Daily Life, and Outlook

    There is no guaranteed way to prevent autoimmune diseases, in part because autoimmune disease causes are still being fully understood.

    However, early recognition of symptoms, attention to family history, and timely medical evaluation can lead to earlier intervention and fewer complications. For those already diagnosed, coordinated care, medication adherence, and healthy routines can make day-to-day life more manageable.

    Living with an autoimmune disease often requires adjustments at work, at home, and in social life. Fatigue, pain, and cognitive difficulties may require pacing, flexible schedules, or accommodations.

    Many people benefit from a support network that includes healthcare providers, mental health professionals, and peer support. Advances in research are improving therapies and deepening understanding of autoimmune disease causes, offering hope for more precise treatments and better long-term outcomes.

    Frequently Asked Questions

    1. Can someone have an autoimmune disease without abnormal blood tests?

    Yes. Some people have clear symptoms and exam findings of autoimmune disease even when early blood tests are normal or borderline. Follow-up testing over time and specialist evaluation are often needed.

    2. Do all autoimmune diseases cause lifelong disability?

    No. Many autoimmune diseases can be managed well with treatment and lifestyle changes. Some people experience long periods with mild symptoms or remission and continue working and staying active.

    3. Are vaccines a common cause of autoimmune diseases?

    Current evidence does not support vaccines as a common cause of autoimmune diseases. Infections themselves are more strongly linked to triggering autoimmunity than vaccination.

    4. Can changing diet alone reverse an autoimmune disease?

    Diet changes may reduce symptom severity and inflammation for some individuals, but they typically do not replace medical treatment. Food choices work best as part of a broader care plan, not as the only therapy.



    Source link

  • When an Autoimmune Skin Condition Signals Systemic Inflammation

    When an Autoimmune Skin Condition Signals Systemic Inflammation

    Psoriasis is often seen as a surface-level issue, but the red, scaly plaques on the skin can be a visible sign of a deeper inflammatory process. As understanding of this autoimmune skin condition has grown, researchers now recognize that psoriasis symptoms may reflect widespread immune activity affecting joints, blood vessels, and metabolism.

    Viewing psoriasis through this systemic lens can change how people think about diagnosis, treatment, and long-term health.

    Understanding Psoriasis as an Autoimmune Skin Condition

    Describing psoriasis as an autoimmune skin condition highlights the role of the immune system in driving the disease.

    In autoimmune and immune-mediated conditions, the body’s defenses become misdirected, leading to chronic inflammation even in the absence of infection. In psoriasis, this inflammation focuses on the skin and, in some people, the joints.

    Some experts refer to psoriasis as both autoimmune and autoinflammatory because it involves elements of classic autoimmunity and innate immune activation.

    Regardless of terminology, the key idea is that psoriasis is not caused by poor hygiene or simple irritation. It is a biologically complex condition with genetic and environmental contributors, and its symptoms reflect immune pathways that can also affect other organs.

    Recognizing Common Psoriasis Symptoms

    Psoriasis symptoms can look different from person to person, but several features are particularly characteristic. The most common form, plaque psoriasis, typically presents with:

    • Raised, thickened patches of skin covered with silvery-white or grayish scales
    • Red, pink, or purple discoloration, depending on skin tone
    • Lesions most often found on the elbows, knees, scalp, and lower back

    Many individuals report itching, burning, or soreness in affected areas, which can interfere with sleep and daily activities. The skin may become dry and cracked, with occasional bleeding when plaques are irritated or scratched. The scalp can show heavy flaking that resembles severe dandruff.

    Psoriasis symptoms are not limited to the skin. Nail changes are common, including pitting (small depressions), ridging, discoloration, and separation of the nail from the nail bed.

    Different subtypes—such as guttate, pustular, inverse, and erythrodermic psoriasis—have their own patterns of symptoms, ranging from small drop-like spots to widespread redness or pus-filled bumps. This diversity is one reason why a professional evaluation is important when symptoms first appear.

    How Psoriasis Connects to Systemic Inflammation

    The connection between psoriasis and systemic inflammation lies in the immune pathways that drive both skin lesions and deeper health effects. Immune cells in psoriatic plaques produce cytokines—chemical messengers that promote inflammation.

    These cytokines do not remain confined to the skin; they circulate in the bloodstream, potentially affecting the lining of blood vessels, joints, and other tissues, according to Mayo Clinic.

    This understanding supports the view of psoriasis as part of a broader inflammatory network that can raise the risk for other conditions. In this sense, psoriasis symptoms on the skin can serve as a visible marker of underlying immune activity that may be occurring silently elsewhere in the body.

    Recognizing this connection has encouraged more comprehensive care, with attention not only to skin clearance but also to overall inflammatory burden.

    Triggers That Worsen Psoriasis Symptoms and Inflammation

    Psoriasis symptoms often fluctuate over time, with periods of relative calm and sudden flares. A variety of triggers can worsen the condition, including:

    • Infections, particularly streptococcal throat infections in guttate psoriasis
    • Skin injuries such as cuts, scrapes, or sunburn (the Koebner phenomenon)
    • Psychological stress and lack of sleep
    • Certain medications, including some blood pressure drugs and antimalarials
    • Smoking and heavy alcohol use

    Lifestyle factors also play a major role. Obesity and inactivity can promote systemic inflammation and increase disease severity. Dietary patterns high in processed foods and low in fruits, vegetables, and whole grains may contribute to an inflammatory state.

    Genetics and family history further influence who develops psoriasis and how severe it becomes, highlighting the interplay between inherited risk and environmental exposures.

    Diagnosis and Assessment of Systemic Involvement

    Diagnosis of psoriasis is usually clinical, based on the appearance and distribution of lesions and the presence of characteristic scaling.

    Dermatologists may perform a skin biopsy in uncertain cases to confirm the diagnosis and rule out other conditions. Because psoriasis is linked with systemic inflammation, many experts also recommend screening for comorbidities.

    A thorough assessment may include questions about joint pain, stiffness, and swelling to identify psoriatic arthritis. Blood pressure, weight, waist circumference, and basic blood tests can help evaluate cardiovascular and metabolic risk.

    In some cases, referral to rheumatology or other specialists is appropriate. Approaching psoriasis in this integrated way supports early detection of related conditions and more comprehensive management.

    Treatment Options for Skin and Systemic Inflammation

    Treatment strategies for psoriasis aim to relieve symptoms, improve skin appearance and function, and reduce the underlying inflammatory activity. For mild disease, topical therapies are often the first line.

    These include corticosteroid creams and ointments, vitamin D analogues, retinoids, calcineurin inhibitors, and other medicated products that reduce inflammation and slow skin cell growth, as per Cleveland Clinic.

    For more extensive or resistant psoriasis, phototherapy (controlled exposure to ultraviolet light) can be effective. Systemic medications such as methotrexate, cyclosporine, and newer oral agents are used to modulate immune responses in moderate to severe cases.

    Biologic therapies have become a major advance; these drugs target specific molecules such as interleukin-17, interleukin-23, or tumor necrosis factor-alpha. By focusing on key inflammatory pathways, biologics can improve both skin and joint symptoms and may help lower overall inflammatory burden.

    The choice of treatment depends on disease severity, comorbid conditions, patient preferences, and safety considerations. Regular follow-up is important to monitor effectiveness, side effects, and long-term health.

    Psoriasis Symptoms as a Clue to Whole-Body Health

    When psoriasis is viewed only as a cosmetic concern, opportunities to protect long-term health can be missed. Recognizing it as an autoimmune skin condition that reflects broader immune activity changes the conversation.

    Skin lesions, nail changes, and joint symptoms can act as visible clues to underlying systemic inflammation, providing an early chance to identify and address associated risks.

    People living with psoriasis benefit from collaborative care that includes dermatology, primary care, and when appropriate, rheumatology or cardiology. Open communication about new or changing symptoms, treatment goals, and lifestyle challenges helps tailor a plan that addresses both the skin and the whole person.

    By integrating medical therapy with attention to cardiovascular, metabolic, and joint health, psoriasis can be managed not just as a skin disease but as a key window into overall inflammatory wellness.

    Frequently Asked Questions

    1. Can someone have psoriasis without the classic red, scaly plaques?

    Yes. Psoriasis can appear as smooth red patches in skin folds, only affect the nails, or show mainly as joint pain with minimal visible skin changes.

    2. If psoriasis is an autoimmune skin condition, does that mean the immune system is “weak”?

    No. The immune system is overactive and misdirected, creating chronic inflammation, not too weak to fight infections.

    3. Can psoriasis go into remission, and what does that look like?

    Yes. Plaques can flatten or disappear, itching can resolve, and skin may look almost normal, though flares can return.

    4. How is psoriasis different from eczema if both cause itchy, inflamed skin?

    Psoriasis usually forms thick, sharply edged, scaly plaques and is strongly tied to systemic inflammation; eczema tends to be less defined, may ooze, and is more linked to allergies and a weak skin barrier.



    Source link

  • Key Autoimmune Arthritis Warning Signals

    Key Autoimmune Arthritis Warning Signals

    Rheumatoid arthritis (RA) is a chronic autoimmune arthritis that affects millions worldwide. Recognizing the early signs and symptoms is crucial for timely treatment and preventing joint damage.

    This article explores the early rheumatoid arthritis symptoms, typical RA signs, how the disease differs from other autoimmune arthritis conditions, and when to see a doctor for evaluation.

    What Is Rheumatoid Arthritis?

    Rheumatoid arthritis is an autoimmune arthritis condition where the immune system mistakenly attacks the body’s own joint tissues. Unlike osteoarthritis, which results from wear and tear, RA causes inflammation inside the joint lining that leads to swelling, pain, stiffness, and eventual joint destruction. It most commonly affects small joints such as those in the hands, wrists, and feet, but it can progress to larger joints as well.

    RA is systemic, meaning it can also affect other organs in the body, including the heart, lungs, and eyes. While the exact cause remains unclear, genetic predisposition and environmental factors like smoking increase the risk of developing this autoimmune disorder.

    What Are the Early Rheumatoid Arthritis Symptoms?

    Identifying early rheumatoid arthritis symptoms is vital as immediate medical attention can slow the disease’s progression. The primary early RA signs include:

    • Joint Pain and Stiffness: Inflammation causes the joints to ache, especially after periods of inactivity. This pain is often symmetrical, affecting joints on both sides of the body.
    • Prolonged Morning Stiffness: One of the classic autoimmune arthritis symptoms is stiffness that lasts for more than an hour in the morning or after resting. This is different from other arthritis types where morning stiffness is typically brief.
    • Joint Swelling and Redness: Inflamed joints often appear swollen, warm, and sometimes red. These signs indicate active inflammation.
    • Fatigue and Malaise: General feelings of tiredness and low energy often accompany the joint symptoms. This systemic symptom is common in RA due to widespread inflammation.
    • Low-grade Fever: Some individuals may experience a mild fever, signaling the autoimmune activity in the body.

    These early symptoms may be subtle, making it important to monitor changes carefully.

    How to Recognize RA Signs in Daily Life

    Early RA signs can interfere with daily activities. People may find it difficult to perform routine tasks such as buttoning shirts, opening jars, or walking comfortably because of joint stiffness, according to the World Health Organization. Noticing symmetrical joint discomfort, for example, pain in both hands or both wrists, can be a key indicator of RA rather than mechanical injury or osteoarthritis.

    The combination of joint symptoms with systemic complaints like fatigue or unexplained low-grade fever should raise suspicion for autoimmune arthritis and prompt medical evaluation.

    Which Joints Are Affected First in Rheumatoid Arthritis?

    RA typically targets the small joints first, particularly those in the hands, wrists, and feet. The joints around the knuckles (metacarpophalangeal joints) and the base of the fingers are commonly involved early. Unlike osteoarthritis, which often affects the thumbs and large weight-bearing joints, RA’s inflammation is characteristically symmetrical and persistent.

    This pattern helps distinguish RA from other arthritis types. Recognizing which joints are affected early on supports faster diagnosis and treatment initiation.

    How Is Early Rheumatoid Arthritis Diagnosed?

    Diagnosing RA in its early stages involves a combination of clinical evaluation, laboratory testing, and imaging. A healthcare provider will assess for RA signs such as swollen, tender joints and review the duration and pattern of joint symptoms, as per the Centers for Disease Control and Prevention.

    Laboratory tests are essential to confirm the diagnosis:

    • Rheumatoid Factor (RF): An antibody present in many but not all RA patients.
    • Anti-Cyclic Citrullinated Peptide (anti-CCP) Antibodies: Highly specific for RA and often present even before symptoms develop.
    • Inflammatory Markers: Elevated erythrocyte sedimentation rate (ESR) or C-reactive protein (CRP) indicate active inflammation.

    Imaging techniques such as X-rays can detect joint erosion, but changes may not appear early. Ultrasound and MRI are more sensitive and can identify inflammation and damage before it is visible on X-rays.

    Early diagnosis is critical because starting disease-modifying antirheumatic drugs (DMARDs) early reduces joint damage and improves long-term outcomes.

    What Are the Differences Between Rheumatoid Arthritis and Other Autoimmune Arthritis?

    While RA is a common form of autoimmune arthritis, it is distinct from other autoimmune diseases like lupus or psoriatic arthritis.

    • Lupus: Often affects skin, kidneys, and blood cells with joint symptoms but usually less severe joint erosion.
    • Psoriatic Arthritis: Associated with psoriasis skin lesions and affects joints asymmetrically.

    Some RA signsspecifically include symmetrical joint inflammation, presence of anti-CCP antibodies, and particular patterns of joint involvement.

    Understanding these differences aids in accurate diagnosis and tailored treatment approaches.

    When Should You See a Doctor for Rheumatoid Arthritis Symptoms?

    Prompt medical consultation is recommended when experiencing persistent joint pain, swelling, and stiffness lasting more than six weeks, especially if these symptoms involve multiple joints on both sides of the body.

    Early signs such as prolonged morning stiffness and unexplained fatigue coupled with joint discomfort are also red flags. Since RA can cause irreversible joint damage, early intervention by a rheumatologist significantly improves prognosis.

    How Can Early Rheumatoid Arthritis Be Managed?

    Effective early management focuses on controlling inflammation to prevent joint damage and improve quality of life.

    Disease-Modifying Antirheumatic Drugs (DMARDs): Medications like methotrexate reduce immune system activity and slow progression.

    • Biologic Therapies: Target specific immune system components to suppress inflammation.
    • Physical Therapy: Helps maintain joint flexibility and muscle strength.
    • Lifestyle Adjustments: Regular exercise, balanced diet, and quitting smoking support treatment effectiveness.

    Early, aggressive treatment strategies improve symptoms and reduce long-term disability associated with RA.

    Frequently Asked Questions

    1. Can rheumatoid arthritis affect people of all ages?

    While RA is more common in middle-aged adults, it can affect individuals of any age, including children (known as juvenile idiopathic arthritis) and the elderly. Age does not exclude anyone from developing this autoimmune arthritis.

    2. Are there specific lifestyle factors that can trigger rheumatoid arthritis flare-ups?

    Certain factors like stress, infections, smoking, and poor diet may exacerbate inflammation and trigger RA flare-ups. Managing these triggers alongside medical treatment can help reduce symptom severity.

    3. How do gender and genetics influence the risk of developing RA?

    Women are two to three times more likely to develop rheumatoid arthritis than men, likely due to hormonal and genetic influences. A family history of autoimmune arthritis also increases individual risk.

    4. What role does diet play in managing autoimmune arthritis symptoms?

    While no specific diet cures RA, anti-inflammatory foods such as omega-3 rich fish, fruits, vegetables, and whole grains may help reduce inflammation and support overall joint health when combined with medical treatment.



    Source link

  • Does Fasting Help Autoimmune Diseases? 

    Does Fasting Help Autoimmune Diseases? 

    Various fasting regimens have been attempted for inflammatory autoimmune diseases, such as lupus, ankylosing spondylitis, chronic urticaria, mixed connective-tissue disease, glomerulonephritis, and multiple sclerosis, as well as osteoarthritis and fibromyalgia.

    The strongest evidence of the benefits of fasting surrounds the treatment of rheumatoid arthritis, an autoimmune joint disease, as I detailed in my previous blog post. A German study suggested benefits for osteoarthritis, too, and reported improvements in pain and joint function, but we’d really need randomized controlled studies to know for sure. The researchers despaired they only had 30 patients, but that’s 30 times more than many reports on fasting in the medical literature, which may detail only single cases.

    One woman, for example, with a rare autoimmune disease known as mixed connective-tissue disease, which can cause all sorts of painful and distressing symptoms, was treated with steroids in an attempt to suppress her immune system. After 21 days of fasting, and off her medications, “she had no further complaints.” More importantly, her symptoms seemed to stay away, and “she remained free of medication.” So, does fasting work for mixed connective tissue disease? All we can say is that it worked at least once.

    A similar success story was reported with fibromyalgia. A woman with pain throughout her body, who couldn’t sustain activity and was on a lot of drugs, became “symptom-free” after a 24-day fast and remained that way at her “follow-up visit 1 month later.” However, when a modified fasting regimen was tried on dozens of individuals, the benefits seen at week 2 largely disappeared by week 12, as shown below and at 1:32 in my video Fasting for Autoimmune Diseases. 

    What about lupus? A 45-year-old woman who had remained in pain despite taking immunosuppressive drugs was pain-free by day four of fasting and remained symptom-free for one year when her symptoms began to recur, but she wiped them out again with a second seven-day fast, “after which she had no symptoms.” Note, though, that she didn’t only fast. She water-only fasted, then followed it with a plant-based diet in an attempt to solidify the gains. On its own, a strictly plant-based diet without any animal protein has been shown to control symptoms in at least some cases.

    The same with sacroiliitis, a common manifestation of ankylosing spondylitis, an autoimmune arthritis that primarily affects the spine, causing back pain that can last for years. In the case of a 33-year-old man, all sorts of conventional therapies and drugs were tried, but the pain wouldn’t go away. So, the complete avoidance of animal foods was recommended, and “the complaints improved distinctly and persistently” within days—until the patient ate meat again. Once again, back on plant-based nutrition, he was off most of his drugs and almost completely free of symptoms. So, at least in this case, inflammatory pain refractory to other treatments was abolished by eating more healthfully. At least it’s worth a try!

    Autoimmune glomerulonephritis, where your body attacks your own kidneys, is a common manifestation of lupus. In a case series, 29 patients were fasted for 60 hours, then had only fruits and vegetables until they got better. They described such remarkable recoveries that fasting, in their opinion, “should be an essential part of treatment.”

    What about multiple sclerosis, an autoimmune nerve disease? Individuals with MS were randomized to a “fasting-mimicking diet”—in this case, a modified fast that started out with an 800-calorie-a-day diet of fruit, rice, or potatoes, followed by a week sipping a few hundred calories of flaxseed oil and vegetable broth, then transitioning to a plant-based Mediterranean diet. Over the next three months, they experienced a significant improvement in overall quality of life. They also tried a ketogenic diet, but that failed to offer clinically or statistically significant overall benefit, as you can see below and at 3:34 in my video. 

    And, finally, let’s look at chronic urticaria (hives), where you get a rash of itchy weals and welts, as seen here and at 3:40 in my video. Individuals started to improve on day 3 of the fast, and their hives completely disappeared by day 11. This is consistent with studies from Germany and Japan that evidently showed around 75 percent effectiveness for such patients with what looks like some sort of tea with sugar diet. It’s certainly worth giving fasting therapy a try, but, of course, fasting should only be done under trained medical supervision. Otherwise, you’d never know if you have some hidden underlying kidney issue that could land you in a coma, then maybe in the morgue. You have to have your kidney function and electrolytes monitored to make sure your body is up for the challenge. 

    “Despite the possible good outcomes, water-only fasting is not a cure or treatment in the traditional sense; it is simply intended to promote the body’s self-healing mechanisms.” Since fasting is unsustainable, by definition, “to maintain the results obtained by water-only fasting, it is necessary to adhere to a health-promoting lifestyle that includes a diet of minimally processed plant foods, adequate sleep, and robust physical exercise.”

    If you haven’t seen it yet, check out my related video Fasting for Rheumatoid Arthritis.

    I’ve held three webinars on fasting. All of the videos are available for free on NutritionFacts.org, but you can also get them in a digital download—as a bundle or separately. See: 

    To see all of the fasting videos currently on the site, please visit our fasting topic page.

    Interested in more on using diet to prevent and treat autoimmune diseases? Check out the related posts below.



    Source link

  • Vitamin D Deficiency In Kids Raises Risk Of Auto-Immune Disorders: Study Explains How

    Vitamin D Deficiency In Kids Raises Risk Of Auto-Immune Disorders: Study Explains How

    Ensure your child is getting adequate sunshine vitamins. Beyond the role of calcium absorption and bone strengthening, researchers have discovered more about its connection to the immune system. A recent study revealed how a deficiency of vitamin D in young children can raise the risk of developing autoimmune disorders.

    According to the latest study published in the journal Science Advances, the deficiency of vitamin D in kids is linked to premature aging of the thymus gland. The thymus is a small lymph gland that makes and trains T-cells that are crucial for the immune system, defending the body against infections and diseases. While most of these vital cells are produced before birth, the remainder are generated during childhood, and by puberty, individuals would have developed the complete T cells for their lifetime.

    “An aging thymus leads to a ‘leaky’ immune system. This means the thymus becomes less effective at filtering out immune cells that could mistakenly attack healthy tissues, increasing the risk of autoimmune diseases like type 1 diabetes,” said the lead author of the study John White in a news release.

    Studies have shown that vitamin D is essential for immunity but according to White, the recent study findings “bring new clarity to this connection and could lead to new strategies for preventing autoimmune diseases.”

    During the trial, researchers examined mice that could not produce vitamin D to assess how this deficiency affected their thymus glands. They then conducted cell analyses and gene sequencing to understand the impact of vitamin D deficiency on the immune system. The researchers expect similar findings in humans as the thymus functions similarly in both species.

    Based on the study findings, researchers suggest that young children should get adequate vitamin D, either through natural sources such as fortified food and sunlight or through supplementation.

    “If you have a young child, it’s important to consult with your healthcare provider to ensure they’re getting enough,” said White.

    Earlier research conducted by the same team in 2021, which followed up more than 10,000 children showed that early vitamin D supplementation can reduce the risk of developing type 1 diabetes by up to five-fold.

    Source link